- (A) Respiratory alkalosis
- (B) Haemorrhage
- (C) Hepatic necrosis
- (D) Gastric Ulceration
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- (A) Reye’s syndrome
- (B) Nausea
- (C) Diarrhoea
- (D) Acid-base imbalance
- (A) Analgesia is associated with μ and k receptors
- (B) Dysphoria is associated with S receptors
- (C) NSAIDS benefit by preventing prostaglandin synthesis
- (D) Naloxane is a non-competitive antagonist and irreversibly opposes the opioids
- (A) Remifentanil
- (B) Morphine
- (C) Sufentanil
- (D) Fentanyl
- (A) Naloxone
- (B) Nalosphine
- (C) Nalpuphine
- (D) Methadine
- (A) It inhibits thromboxanes
- (B) It stimulates Prostacyclins
- (C) It is a vasodilator
- (D) Ithelps in reducing inflammatory aggregate
- (A) Vitamin K
- (B) Barbiturates
- (C) Acetominophen
- (D) Acetyl salicylic acid
- (A) Agranulocytosis
- (B) Hemolytic anemia
- (C) Aplastic anemia
- (D) Hypoprothrombinemia
- (A) Prostaglandin synthesis
- (B) Ectoglandin synthesis
- (C) Preglandin synthesis
- (D) Costaglandin synthesis
- (A) Less thrombophlebitis due to low solubility in blond
- (B) More plasma binding and less toxicity
- (C) More potent than diazepam
- (D) Binds to benzodiazepine receptors with 3-4 time more avidly
